As you continued to experiment with higher concentrations of the toxin, you found cases when the cell could not repolarize at all, or if it began to repolarize, it would immediately depolarize again. Given this description and the description in the previous question, how does this toxin acts on voltage-gated sodium ion channels? The toxin probably prevents the potassium channels from opening. This would cause the cell to depolarize properly but not repolarize since the potassium channels are needed for that to occur. The toxin probably prevents the sodium channels from opening. The cell would not be able to depolarize properly if the sodium channels are blocked. The toxin probably prevents the sodium channels from closing once they are opened. This would cause the cell to depolarize properly at first, but not repolarize since the sodium channels need to close for that to occur. The toxin probably prevents the sodium channels from opening. The cell would not be able to repolarize properly if the sodium channels are blocked. none of these