Answer:
Blood clotting normally occurs when there is damage to a blood vessel.
Explanation:
Platelets immediately begin to adhere to the cut edges of the vessel and release chemicals to attract even more platelets. A platelet plug is formed, and the external bleeding stops.
Next, small molecules, called clotting factors, cause strands of blood-borne materials, called fibrin, to stick together and seal the inside of the wound. Eventually, the cut blood vessel heals and the blood clot dissolves after a few days.
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Answer:
I think you mean blood clotting so umm
Explanation:
Blood is made up of many different cells—mostly red blood cells, white blood cells and platelets—as well as clotting factors. These and other elements float through blood vessels in a strawlike liquid called plasma.
The coagulation cascade is a complex chemical process that uses as many as 10 different proteins (called blood clotting factors or coagulation factors) that are found in plasma. Put simply, the clotting process changes blood from a liquid to a solid at the site of an injury. Here’s how the process works:
Injury. A cut on the skin or an internal injury creates a small tear in a blood vessel wall, which causes blood flow.
Vessel constriction. To control blood loss, the blood vessel immediately narrows (called constriction), which limits blood flow through the vessel.
Platelet plug. In response to the injury, tiny cells in the blood called platelets are activated. The platelets stick to one another and to the wound site to form a plug. The protein von Willebrand factor (VWF) helps the platelets stick to each other and to the blood vessel wall.
Fibrin clot. Next, blood clotting factors trigger production of fibrin, which is a strong, strandlike substance that surrounds the platelet plug and forms a fibrin clot, a meshlike net that keeps the plug firm and stable. Over the next several days to weeks, the clot strengthens and then dissolves as the wounded blood vessel wall heals.
