A Final statement or concluding statement
It should be noted that not all gut microbiota promote fatty liver disease; some strains act as "probiotics," mitigating the negative effects of other gut microflora.
This is supported by evidence that butyrate, as demonstrated in NAFLD rat models, may be the mechanism by which some bacteria inhibit progression from benign hepatic steatosis to NASH. Changes in the microbiome may thus influence an individual's risk of developing NAFLD, progressing to NASH, or developing the metabolic syndrome.
Hepatic de novo lipogenesis (DNL) is the biochemical process of synthesising fatty acids from acetylCoA subunits produced by a variety of cell pathways, most notably carbohydrate catabolism.
In addition to glucose, which is the most common source of carbon units for DNL, fructose is a profoundly lipogenic substrate that can drive DNL, which is significant given the increased use of fructose in corn syrup as a sweetener.
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