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When acetylcholine binds to its receptor, the muscle is stimulated and contracts. In myasthenia gravis, immune proteins called antibodies block, alter, or even destroy acetylcholine receptors at the neuromuscular junction, preventing the muscle from contracting.
How does myasthenia gravis develop in relation to the immune system?
Myasthenia gravis causes antibodies to be produced by your immune system that either block or harm many acetylcholine receptor sites in your muscles. Because there are fewer available receptor sites, your muscles get fewer nerve impulses, which results in weakening.
Myasthenia gravis (MG) is a long-term autoimmune disorder in which antibodies prevent the nerves' ability to communicate with the muscles, weakening the skeletal muscles as a result. Acetylcholine receptor antibodies are the main cause of myasthenia gravis, a neurological disorder that weakens and exhausts the voluntary muscles.
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